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![]() | Baltic Sea Network on Occupational Health and Safety | |||
| Act on Occupational Diseases 1343/88Issued in Helsinki, 29 December l988
What is stated in the first subsection about occupational diseases, shall also be applied to notable worsening of another disease or injury than occupational during the period of this deterioration.
It can be stated by ordinance that the causal connection between the disease mentioned in the first subsection of Paragraph 1 and a physical, chemical or biological factor in work is regarded as existing when such a factor has been present in the work in such an amount that it principally can cause the disease designated by the Act.
Liability for compensation, the amount of compensation payable, and the procedure to be followed for that purpose shall be governed by the Accident Insurance Act, the Act on Agricultural Workers' Accident Insurance, and the Act entitling persons employed in a public service of holding public office to compensation in the event of accident. For this purpose the date on which the disease manifests itself shall be equated with the date of occurrence of the accident. If other specific reasons do not require it, the date of manifestation of the disease shall be determined as the date when a person for the first time has sought medical advice from a doctor concerning a later diagnosed occupational disease. The time limit within which compensation must be claimed shall invariably be reckoned as beginning on the date on which the disease is diagnosed or the incapacity of the person begins. When a worker, agricultural entrepreneur or person employed in a public service or holding public office is not on the appearance of an occupational disease, engaged in a process that could have been the cause of the disease, liability for compensation shall be determined on the basis of the employment, agricultural entrepreneurship, public service or public office in which he was last engaged in a process that could have been the cause of the disease.
An Ordinance can state more precisely: 1) the determination of the disease and the factors exposing to it; 2) the liability for compensation in the case of tenovaginitis and humeral epicondylitis; 3) the other measures of execution of this Act.
A copy of this Act and the ordinance made thereunder shall be posted and kept available by the employer at the workplace.
This Act shall come into force on 1 January 1989 and repeal the Act on Occupational Diseases (638/67) and its later modifications.
Ordinance on Occupational Diseases 1347/88Issued in Helsinki, 29 December 1988
Diagnosis of a disease as an occupational disease requires such medical examination where there is sufficent knowledge about exposure in the work and where in the case of occupational diseases designated by the Act on Occupational Diseases in Paragraph 2 a specialist in the field is in charge.
A disease shall be deemed as occupational according to 2 § and the first subsection of 4 § and later in 3 §, when the physical, chemical or biological factor mentioned in the paragraph is present in a person's work, and is covered by subsection 1 of 1 § in the Act on Occupational Diseases, to such an extent that its exposure effect is sufficient to cause the disease in question, unless it is stated that the disease has been clearly caused by exposure outside work.
The following are the diseases and the physical, chemical and biological factors referred to in Paraqraph 2:
Typical forms of disease White finger syndrome; polyneuropathy of the upper limb.
Typical forms of disease Cochlear type of deterioration of hearing
Typical forms of disease Direct effects of changes of pressure, such as maxillary hemorrhages and tympanal ruptures; indirect effects of pressure such as nitrous inebriation and diver's disease; as a long-term effect an aseptic bone necrosis of big joints.
Typical forms of disease Bone marrow injuries, lens opacities, skin changes (eczemas, wounds, scars, skin cancer)
Typical forms of disease Lens opacities, e.g. glassblower's cataract; skin changes (connective tissue chanqes, telangiectasies).
Typical forms of disease Conjunctivitis and keratitis of the eye; skin changes (light eczema, light contact eczema).
Typical forms of disease Acute arsenic intoxication (gastro-intestinal, respiratory, and nervous symptoms); long-term respiratory, mucous membrane symptoms; conjuctival irritation of the eye; skin changes like chronic eczema, skin pigmentation, hyperkeratosis, skin cancer; pulmonary cancer; peripheral neuropathies.
Typical forms of disease Irritation of mucous membranes; chemical pneumonitis in high exposure; chronic berylliosis; skin changes (contact eczema, foreign body reaction e.g. granuloma); pulmonary cancer.
Typical forms of disease Irritation of mucous membranes and gastro-intestinal tract in acute intoxication, sometimes chemical pneumonitis. In subchronic or chronic intoxication the symptoms vary according to individual factors and form of exposure: symptoms of the mouth (gingivitis), peripheral and central nervous injuries (e.g. shake, psychic changes, renal injuries (albuminuria) and in connection with the injuries, elevated mercury levels in urine and blood; skin changes (contact eczema, eczema or other wide-spread rash).
Typical forms of disease Injuries of bone and liver; respiratory irritation; central nervous symptoms; caustic injuries of the skin; depression of cholinesterase activity of the tissues in organic phosphorous compund intoxications
Typical forms of disease Acute intoxication with strong respiratory symptoms (chemical pneumonitis); chronic intotication (renal injuries, emphysema); skin changes (contact eczema); pulmonary cancer.
Typical forms of disease Skin changes (contact eczema); rhinitis and asthma due to cobalt allergy; hard metal lunq.
Typical forms of disease Local dermatic or mucosal irritation or corrosion caused by chromium (chrome wounds); skin changes (contact eczema); rhinitis and asthma due to chromium compound allergy; pulmonary cancer; sinusal cancer.
Typical forms of disease The first sign of subchronic or chronic inorganic lead intoxication is disturbed hemoglobin synthesis, later anemia, reticulocytosis, peripheral nerve injuries, gastrointestinal symptoms, liver and kidney injuries, and central nervous symptoms. Organic lead intoxication is characterized by central nervous symptoms. In inorganic lead intoxication symptoms are associated with elevated blood lead level and elevated erythrocyte protoporphyrin values, and in organic lead intoxication elevated lead levels in blood and urine.
Typical forms of disease Acute chemical pneumonitis; chronic manganese intoxication (manganism), dominated by nervous symptoms.
Typical forms of disease Skin changes (contact eczema); rhinitis and asthma due to nickel allergy; chemical pneumonitis caused by nickel carbonyl; sinusal and pulmonary cancer.
Typical forms of disease Zinc fever; skin changes caused by zinc chloride (contact eczema, corrosion).
Typical forms of disease Irritation of respiratory tract (chemical pneumonitis, bronchial constriction).
Typical forms of disease Irritation and corrosion of mucous membranes and conjunctiva; chemical pneumonitis; bone changes caused by fluorine compounds (fluorosis); fever caused by fluorine polymer dispersion products (polymer fever); skin changes (contact eczema, corrosion caused by fluorides).
Typical forms of disease Acute cyanide intoxication, chronic intoxication (respiratory symptoms, nervous symptoms); respiratory diseases caused by isocyanates (asthma).
Typical forms of disease Acute intoxication with mainly central nervous symptoms; chronic intoxication by carbon disulfide with central and peripheral nervous symptoms, possibly associated with coronary heart disease.
Typical forms of disease Acute intoxications with mainly respiratory and central nervous symptoms, and pulmonary edema.
Typical forms of disease Irritative and inflammatory symptoms of mucous membranes and respiratory organs; corrosion of teeth and eyes; skin chanqes (contact eczema, corrosion).
Typical forms of disease Acute respiratory irritation symptoms; pulmonary edema; local irritation or corrosion of mucous membranes; skin changes (contact eczema, corrosion).
Typical forms of disease Acute intoxication caused by carbon monoxide with mainly central nervous symptoms. The clinical picture is associated with elevation of carbon monoxide hemoglobinemia.
Typical forms of disease Acute irritative symptoms of respiratory tract and conjunctival tissues; pulmonary edema.
Typical forms of disease Skin changes (contact eczema, corrosion); acute irritation or corrosion symptoms of conjunctiva, mucous membranes, respiratory or gastro-intestinal tract.
Typical forms of disease Acute and chronic mainly central and peripheral nervous intoxications; skin changes (contact eczema); leukemias caused by benzene; hemangiosarcoma of the liver caused by vinyl chloride.
Typical forms of disease Acute and chronic mainly nervous system intoxications; skin changes (contact eczema); cardiac arrhythmias and irritative respiratory symptoms caused bs freons.
Typical forms of disease Acute intoxications associated with methemoglobinemia; hemolytic anemia, liver and eye changes caused by trinitrotoluene; skin changes (contact eczema); asthma caused by amines; cancer of urinary bladder caused by aromatic amines.
Typical forms of disease Central nervous and circulatory symptoms (i.e. hypotension, vasodilatation) caused either by acute or by chronic intoxication; skin changes (contact eczema).
Typical forms of disease Skin changes (contact eczema); asthma and rhinitis caused by formaldehyde; acute mainly central nervous intoxications caused by alcohols, ketons, ethers and esters; leukemias caused by ethyleneoxyde.
Typical forms of disease Irritation and corrosion of skin and mucous membranes; asthma and rhinitis caused by acid anhydrides (i.e. phtalic acid, maleinic acid and trimellitinic acid anhydrides).
Typical forms of disease Acute intoxications with respiratory, hepatic, renal and central nervous symptoms; chronic intoxication with central nervous and gastrointestinal symptoms; skin changes (contact eczema, changes in pigmentation); hemolytic anemia; methemoglobinemia; hepatic cancer caused by polychlorinated biphenyls.
Typical forms of disease Skin changes (contact eczema); respiratory allergies.
Typical forms of disease Leukemias, lymphohematopoietic cancers and bladder cancer.
Typical forms of disease Respiratory diseases (asthma, rhinitis); skin changes (contact eczema).
Typical forms of disease Skin changes (contact eczema, contact urticaria, protein contact eczema); allergic rhinitis, conjunctivitis or pulmonary asthma caused by organic dust, Monday fever (byssinosis) caused by raw cotton.
Typical forms of disease Pulmonary diseases caused by quartz and asbestos dust (pneumoconioses); pulmonary cancer and mesothelioma caused by asbestos; consequences of pneumoconioses in respiratory and circulatory organs.
Typical forms of disease Skin changes (contact eczema).
Typical forms of disease Skin changes (contact eczema); asthma and rhinitis caused by reactive dyes.
Typical forms of disease Cancer of liver.
Typical forms of disease Allergic alveolitis; asthma and rhinitis caused by molds; humidifier fever.
Typical forms of disease Different forms of tuberculosis.
Typical forms of disease Hepatitis B, anthrax, erysipelas, ringworm, brucellosis, listeriosis, dermatic mycosis, toxoplasmosis, malaria, bilharziosis.
Tenovaginitis and humeral epicondylitis in subsection two of 4 § in the Act on Occupational Diseases are compensated as occupational diseases caused by a physical factor when caused by performing repetitive, monotonous or strained movements as designated in subsection one of 1 § of the Act on Occupational Diseases.
This ordinance will come to effect as of January 1989. The ordinance (850/48) of 3 December 1948 passed under the Accident Insurance Act and the Act on Occupational Diseases shall remain in force in so far as it relates to occupational diseases.
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